which the authors showed that patients suffering from athero-
sclerotic disease had focal, eccentric lesions compared with
patients with other pathologies who had diffuse and concentric
lesions. We did not observe any association between symp-
tomatic nor with asymptomatic plaques and enhancement.
Intracranial plaque enhancement has been reported inconsis-
tently, and the exact mechanisms are not known because histo-
pathologic validation is lacking.22,26,31 It could be that we have
missed some lesion enhancement, because in some patients no
postcontrast scan was obtained or because most patients were
chronic stroke patients. On the contrary, it may be that lesion