Thus, the growth dynamics of
regularly-fed gut bacteria fits temporally into the dynamics
of host GLP-1 and PYY release, suggesting an inductive role
of gut bacteria, and specifically of E. coli proteins, in mealinduced signaling of intestinal satiety and meal termination.
A differential effect of E. coli proteins from the Exp phase to
stimulate GLP-1, may possibly reflect an incretin role of
GLP-1 in glycemic control