Mutations were revealed in seven st

Mutations were revealed in seven strains in Ser83 codon in gyrA that has already been identified in human strains. Similarly, induction of resistance by a mutation in the remaining gyrB Lys447, which was replaced by arginine,was also founding human strains. Since strains isolated from animals showed the same mutations found in human isolates and the appearance of mutations in response to induction of resistance in susceptible strainsit is suggested that the study be extended to include a large rnumber of isolates,inorder to explain the real effect thattheuseofantibiotics has on the resistance of the strains isolated from animals and the likelihood of a possible transfer of this resistance to humans through food

結果 (中文) 1:

ser83在GYRA的密码子已经被确定在人类株中的7个菌株中揭示了突变。同样地，在余下的gyrB基因lys447，这是由精氨酸取代的突变诱导阻力也创办人的菌株。因为从动物分离株显示了同样的突变在人类分离的外观和突变的在易感strainsit电阻感应建议的研究扩展到包括一个大型的菌株rnumber，序，以解释人类通过食物的真实效果上thattheuseofantibiotics有从动物中分离到的菌株的阻力，这种阻力的可能的转移的可能性

結果 (中文) 2:

七株gyrA Ser83密码已经在人类的菌株鉴定的突变被发现。同样，在剩余的gyrB lys447突变诱导阻力，取而代之的是精氨酸，也是建立人类菌株。由于分离株的动物表现出相同的突变在人类的菌株和响应敏感的strainsit抗性诱导突变的出现是建议的研究扩展到包括一个大的rnumber菌株，为了说明实际效果thattheuseofantibiotics对菌株的耐药性的动物通过食物可能转移的抵抗人类的可能性

結果 (中文) 3:

在中已经确定在人类菌株的 gyrA Ser83 密码子七株显露了突变。同样，诱导的抗诱变中剩余的 gyrB Lys447，取而代之的是精氨酸，还成立人类株。由于分离株动物显示发现在人类株和外观的响应的抵抗的诱导突变易受影响的同一突变 strainsit 被建议研究将扩展到包括分离株，大 rnumber以此来解释真正的效果 thattheuseofantibiotics 已从动物和这种阻力可能转让的可能性对人类通过食物株的抗性