vasa vasora get more abundant, and

vasa vasora get more abundant, and they may also develop in
patients with pathologies like vasculitis, aneurysms, and ath-
erosclerosis. When vasa vasora are the only causative factor
for the enhancement in the intracranial vessel wall, this would
imply that the entire vessel tree will enhance in older patients
and patients with atherosclerosis. We do, however, not observe
abundant enhancement of the intracranial vessels. This may
suggest that different mechanisms are involved in the intracra-
nial vessel wall enhancement.
Asymptomatic and symptomatic lesions were found to have
a different morphological feature. The asymptomatic lesions

結果 (中文) 1:

瓦萨 vasora 得到丰富得多，和他们也可能在发展患者病症如脉管炎、 动脉瘤，和 ath-erosclerosis。当瓦萨 vasora 是唯一的致病因素为提高颅内血管壁，这将意味着整个血管树将提高老年患者与动脉粥样硬化患者。我们这样做，但是，不遵守颅内血管丰富增强。这可能表明不同机制参与 intracra-nial 血管壁增强。无症状与有症状病变被发现有不同的形态特征。无症状性病变

結果 (中文) 3:

瓦萨vasora获得更丰富的，他们也会在
与病变如血管炎、动脉瘤的患者，和ATH—
闭塞。当“瓦萨”号vasora是唯一的致病因素
为了加强在颅内血管壁，这将
意味着整个血管树将加强在老年患者
和动脉粥样硬化患者。然而，我们却没有观察到
丰富的颅内血管增强。这可能
建议不同的机制参与了脑—
NiAl血管壁的增强。
无症状和有症状的病变被发现有
不同形态特征。无症状的病灶