Consistency: the risk increased wit

Consistency: the risk increased with latency, meta-analysis gave in the 10+ years’ latency group OR = 1.62, 95% CI = 1.20–2.19.Specificity: increased risk for glioma was in the temporal lobe.Using meningioma cases as comparison group still increased the risk.Temporality: highest risk was in the 20+ years’ latency group, OR = 2.01, 95% CI =1.41–2.88, for wireless phones. Biological gradient: cumulative use of wireless phones increased the risk. Plausibility: animal studies showed an increased incidence of glioma and malignant schwannoma in rats exposed to radiofrequency (RF) radiation. There is increased production of reactive oxygen species(ROS) from RF radiation. Coherence: there is a change in the natural history of glioma and increasing incidence. Experiment: antioxidants reduced ROS production from RF radiation. Analogy: there is an increased risk in subjects exposed to extremely low-frequency electromagnetic fields. Conclusion. RF radiation should be regarded as a human carcinogen causing glioma.

結果 (繁體中文) 1:

Consistency: the risk increased with latency, meta-analysis gave in the 10+ years’ latency group OR = 1.62, 95% CI = 1.20–2.19.
Specificity: increased risk for glioma was in the temporal lobe.Using meningioma cases as comparison group still increased the risk.
Temporality: highest risk was in the 20+ years’ latency group, OR = 2.01, 95% CI =1.41–2.88, for wireless phones.
Biological gradient: cumulative use of wireless phones increased the risk. Plausibility: animal studies showed an increased incidence of glioma and malignant schwannoma in rats exposed to radiofrequency (RF) radiation. There is increased production of reactive oxygen species
(ROS) from RF radiation. Coherence: there is a change in the natural history of glioma and increasing incidence.
實驗：抗氧化劑降低ROS產生從RF輻射。打個比方：有暴露於極低頻電磁場科目的風險增加。結論。RF輻射應被視為人類致癌物引起神經膠質瘤。

結果 (繁體中文) 2:

一致性：風險隨延遲而增加，元分析在 10 年以上的延遲組 OR = 1.62，95% CI = 1.20*2.19。
特異性：膠質瘤風險增加是在葉葉。使用腦膜瘤病例作為比較組仍然增加了風險。
時間：無線電話的延遲組風險最高，OR = 2.01，95% CI =1.41±2.88。
生物梯度：無線電話的累積使用增加了風險。可合理性：動物研究表明，暴露于射頻（RF）輻射的大鼠患膠質瘤和惡性血小馬的發生率增加。活性氧種類的產量增加
（ROS）來自射頻輻射。一致性：膠質瘤的自然史發生變化，發病率增加。
實驗：抗氧化劑減少了射頻輻射產生的ROS。類比：暴露于極低頻率電磁場的受試者的風險增加。結論。射頻輻射應被視為引起膠質瘤的人類致癌物質。

結果 (繁體中文) 3:

一致性：10年以上潜伏期組的meta分析顯示，風險隨潜伏期新增而新增，OR=1.62，95%CI=1.20-2.19。
特异性：顳葉膠質瘤發病風險新增，以腦膜瘤病例為對照組仍新增發病風險。
暫時性：20年以上潜伏期組的風險最高，或=2.01，95%CI=1.41–2.88，對於無線電話。
生物梯度：累積使用無線電話新增了風險。似是而非：動物研究顯示，暴露於射頻（RF）輻射的大鼠腦膠質瘤和惡性神經鞘瘤的發病率新增。活性氧產生量新增
（ROS）來自射頻輻射。一致性：膠質瘤的自然史發生了變化，發病率也在新增。
實驗：抗氧化劑减少了射頻輻射產生的活性氧。類比：暴露在極低頻電磁場中的受試者風險新增。結論。射頻輻射應被視為人類致癌物引起膠質瘤。