As QUER and QU-CAP have been relate

As QUER and QU-CAP have been related to mechanisms against oxidative stress, we then addressed the question whether these substances have an effect on the main secondary anti-oxidant mechanism of the cell, the proteasome. Young HFL-1 cells were treated with 2 μg/ml QUER, 0.5, 2 and 5 μg/ml QU-CAP or the diluents (0.1% DMSO or 0.1% CAP) for 24 h and the CT-L proteasome activity was measured. The activity was significantly induced following cell treatment with either of the substances. CT-L activity was increased from ~1.5 (for QU-CAP) to ~2.4 folds (for QUER). Moreover we have also detected enhanced protein expression levels of representative proteasome subunits in the same cells and experimental conditions, thus further verifying proteasome activation by QUER and QU-CAP

結果 (繁體中文) 1:

如漆和曲章已涉及到對氧化應激的機制，但我們然後討論了問題，這些物質是否對該儲存格，蛋白酶體主要二級抗氧化機制的影響。年輕 HFL-1 細胞的 2 μ g/m l QUER、 0.5、 2、 5 μ g/m l 曲帽或稀釋劑 (0.1%二甲基亞碸或 0.1%帽) 24 h 和 CT-L 蛋白酶體活性測定。活動是明顯誘導後細胞治療的物質之一。CT-L 活性增高從 ~1.5 (為曲-CAP) 到 ~2.4 褶皺 (為漆)。此外我們也發現代表蛋白酶體亞基增強型的蛋白表達水準在同一細胞和實驗條件，從而進一步驗證蛋白酶體活化的漆和曲章

結果 (繁體中文) 2:

作為曲兒和QU-CAP已涉及到對氧化應激機制，我們則處理問題，這些物質是否對細胞，所述蛋白酶體的主要次級抗氧化劑機理的效果。年輕的HFL-1細胞用2微克/毫升曲兒，0.5％，2和5微克/毫升QU-​​CAP或稀釋劑（0.1％DMSO或0.1％，CAP）24小時處理，在CT-L蛋白酶活性測定。的活性顯著誘導在與任一物質的細胞治療。CT-L活動從〜1.5升高（換曲-CAP）到〜2.4倍（對於曲兒）。此外，我們也發現了代表蛋白酶亞基增強蛋白質表達水平在同一細胞和實驗條件下，從而進一步驗證蛋白酶活化曲兒和QU-CAP

結果 (繁體中文) 3:

作為査詢和qu-cap已對抗氧化應激機制有關，然後我們討論的問題是否這些物質對主要次級抗氧化機制的細胞都有作用，蛋白酶體。年輕HFL-1細胞在2μg/mL査詢，0.5治療，2和5μ克/毫升qu-cap或稀釋劑（0.1% DMSO或0.1%帽）24 h，CT-L蛋白酶體活性測定。的活性顯著誘導後的細胞治療與任一的物質。從~ CT-L活性新增1.5（為qu-cap）到2.4倍（為~査詢）。此外我們還發現在相同實驗條件下細胞和增强代表性蛋白酶體亞基蛋白表達水准，從而進一步驗證通過査詢和qu-cap蛋白酶啟動