which allowsdistinguishing affectivefrom sensorimotor aspectsof emotionalprosody generation. Both experiments showthatwhen
participants prepare for emotional prosody, bilateral ventral striatum is specifically activated and connected to temporal poles and anterior
insula, regions in which lesions frequently cause dysprosody. The bilateral dorsal striatum is more sensitive to cognitive and motor aspects of
emotional prosody preparation and production andismore strongly connectedtothe sensorimotor speech network compared withthe ventral
striatum. Right lateralization during increased prosodic processing is confined to the posterior superior temporal sulcus, a region previously
associated with perception of emotional prosody. Our data thus provide physiological evidence supporting both models and suggest that
bilateralbasalgangliaareinvolvedinmodulatingmotorbehaviorasafunctionofaffective state.Rightlateralizationofcortical regionsmobilized
for prosody control could point to efficient processing of slowly changing acoustic speech parameters in the ventral stream and thus identify
sensorimotor processing as an important factor contributing to right lateralization of prosody.